GE, encodes CYP78A13, is predominantly express in the interfacing tissues of the both embryo and endosperm. GE expression is under negative feedback regulation; endogenous GE expression is upregulated in ge mutants. Loss of function mutant has large embryo and small endosperm, whereas GE overexpression causes a small embryo and enlarged endosperm. A complementation analysis coupled with heterofertilization shows that complementation of ge mutation in either embryo or endosperm failed to restore the wild?type embryo/endosperm ratio (Nagasawa et al., 2013).